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Official Journal of the Italian Society of Orthopaedics and Traumatology

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Management of chronic unstable acromioclavicular joint injuries

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Abstract

The acromioclavicular joint represents the link between the clavicle and the scapula, which is responsible for the synchronized dynamic of the shoulder girdle. Chronic acromioclavicular joint instability involves changes in the orientation of the scapula, which provokes cinematic alterations that might result in chronic pain. Several surgical strategies for the management of patients with chronic and symptomatic acromioclavicular joint instability have been described. The range of possibilities includes anatomical and non-anatomical techniques, open and arthroscopy-assisted procedures, and biological and synthetic grafts. Surgical management of chronic acromioclavicular joint instability should involve the reconstruction of the torn ligaments because it is accepted that from three weeks after the injury, these structures may lack healing potential. Here, we provide a review of the literature regarding the management of chronic acromioclavicular joint instability.

Level of evidence

Expert opinion, Level V.

Introduction

The acromioclavicular joint (ACJ) represents the link between the clavicle and the scapula, which is responsible for the synchronized dynamic of the shoulder girdle [1]. It has been shown that most patients with a history of unstable ACJ injuries managed conservatively develop changes in the anatomical orientation of the scapula, which provokes alterations in the dynamics of the rotator cuff, which can eventually predispose chronic pain [2].

Biomechanical studies have demonstrated the importance of anatomical reconstruction of the coracoclavicular (CC) ligaments in cases of unstable ACJ injuries [3]. This importance lies in the fact that the conoid and trapezoid ligaments have different functions, which depend on their anatomical location and orientation [4].

Many of the procedures for the treatment of unstable ACJ injuries are non-anatomical [5]. The therapeutic approach for chronic ACJ instability should be different from that for acute ACJ instability. In the acute phase, it is accepted that the acromioclavicular (AC) and CC ligaments still have the potential to heal, so surgical techniques may aim to align the ends of the torn ligaments while tissue-healing takes place [6]. On the other hand, as the AC and CC ligaments lose their potential to heal from 3 weeks after the ACJ injury [6], the management of chronic ACJ instability must involve biological augmentation as well as mechanical fixation [7].

Many strategies that have been described for the management of chronic ACJ instability are non-anatomical [8] and lack primary mechanical fixation [9] that protects the graft during integration to the bone.

Here, we present a review of the literature regarding the management of chronic unstable ACJ injuries. As this review is narrative, we only included studies that were found to be of interest in supporting the concepts that we aim to transmit.

Surgical management

Indications for treatment

It is currently accepted that reasonable management for grade III ACJ injuries consists of conservative measures. A second examination (3–6 weeks after shoulder injury) must be carried out to assess the evolution of symptoms. If at 3 months after the shoulder injury (already in chronic phase) a patient with a grade III ACJ injury still complains of symptoms of scapular dyskinesis, and radiographic examinations show overriding of the distal third of the clavicle over the acromion in the Alexander projection, surgical treatment is recommended [10].

Patients with chronic and symptomatic ACJ instability (Rockwood grade III–V) must be informed about the internationally accepted recommendations regarding the surgical treatment of these injuries once the conservative measures have failed. However, they must also be informed about the potential risks of a surgical procedure and about the physical limitations of the postoperative period. In contact players, we initially consider their immediate shoulder requirements, and if they are professional or semi-professional players, we also consider the stage of the season in which they are involved. The indication for surgical treatment in this group of patients must always take the performance expectations of the athlete for the rest of the season into consideration.

Timing for surgery

Weinstein et al. described the time point distinguishing acute versus delayed surgery as 3 weeks after the date of the shoulder injury [6]. In their comparative study, the surgical procedure was the modified Weaver–Dunn technique in 15 of 27 cases managed in the acute setting and in 14 of 17 cases managed in the chronic setting. The rest of the repairs were performed by means of AC non-absorbable sutures. Satisfactory results were obtained in 96% of cases treated in the acute phase and in 76% of cases treated in the chronic phase. The differences were statistically significant in favor of treatment in the acute phase [6].

Rolf et al. compared a group of patients treated immediately after the occurrence of shoulder injury (29 patients, using the modified Phemister technique, adding a CC fixation with sutures) with a group of patients who had undergone surgery after failure of conservative treatment (20 patients using the modified Weaver–Dunn procedure) [11]. The results were significantly superior in the group of patients managed in the acute phase [11].

Mignani et al. compared 25 patients treated in the acute phase with 15 patients treated in the chronic phase [12]. In both groups the management consisted of AC and CC temporary fixations with Kirschner wires and concomitant excision of the distal third of the clavicle. The authors reported satisfactory results in 100% of patients in the acute group and 93% of patients in the chronic group, with no statistically significant differences [12].

Dumontier et al. compared 32 patients treated in the acute phase (first 3 weeks) with 24 patients treated in the chronic phase (>3 weeks) [13]. All patients were treated by means of transposition of the coracoacromial (CA) ligament. The results were satisfactory in 81% of patients treated in the acute phase and in 79% of patients treated in the chronic phase [13]. The study reported no significant differences between groups.

Von Heideken et al. compared 22 patients treated in the acute phase (within the first 4 weeks after injury) with 15 patients treated in the chronic phase (after a minimum of 4 months of conservative measures) [14]. The technique used was ACJ fixation with a hook plate. The results were significantly superior, both in the clinical and radiological aspects, in the group of patients managed in the acute phase [14]. A summary of the main aspects of these studies is shown in Table 1.

Table 1 Management in the chronic setting versus management in the acute setting

Surgical techniques for the management of chronic ACJ instability

Coracoacromial ligament transposition

The most classical method for the surgical management of chronic ACJ instability is the technique that involves transposition of the CA ligament (Fig. 1) [15, 16]. The technique described by Weaver and Dunn involves excision of the distal third of the clavicle, detachment of the AC ligament from the acromion, and transposition of this ligament to the distal third of the clavicle [16]. The modifications made to the original Weaver–Dunn procedure aimed to increase the primary mechanical stability of the fixation, by means of adding a CC fixation with subcoracoid suture loops [17], coracoid suture anchors [18], or tendon grafts. Another described modification consists of the addition of a hook plate [19].

Fig. 1
figure1

Superolateral intraoperative perspective of a left shoulder with a history of chronic ACJ dislocation, that was managed by means of a modified Weaver–Dunn procedure. a Visualization of the coracoacromial (CA) ligament previous to its transfer to the distal third of the clavicle. Sutures have already been passed through the bone tunnels. The most medial tunnel aimed to achieve coracoclavicular (CC) fixation. This suture was previously passed beneath the coracoid process. b Details of the final suture fixation. Sutures are passed through the bone tunnels created in the clavicle

Boileau et al. described an all-arthroscopic Weaver–Dunn–Chuinard procedure with double-button fixation for chronic ACJ dislocations [15]. The authors performed the above-mentioned technique in 10 consecutive patients with ACJ injuries (Rockwood type III or IV). After a mean follow-up of 12.8 months, the authors reported that patients were satisfied or very satisfied with the cosmesis; 9 of 10 patients returned to previous sports, and all symptoms resolved in all patients. They concluded that the bone block transfer (Weaver–Dunn–Chuinard procedure) involves the advantage of being a stronger repair, providing bone-to-bone healing by using free, autologous, vascularized tissue [15]. The authors reported that double-button fixation has the advantage of maintaining the reduction during the biological healing process. We believe that this technique involves a biomechanical disadvantage related to the transposition of the CA ligament [20].

Studies have shown the inferior characteristics of the CA ligament compared to the native ACJ [20]. The clinical outcomes obtained by means of the described modifications to the Weaver–Dunn technique have been described as satisfactory [1719]. However, it is noteworthy that the use of the hook plate has been associated with a higher rate of complications, including infection, plate dislocation and need for re-operation [19]. Coracoid suture anchors have been associated with a higher rate of secondary displacements [18].

Two of the modifications made to the Weaver–Dunn technique have been compared (CC fixation with PDS vs hook plate) [17]. Clinical results were similar between groups, but the authors stated that the advantage of CC fixation with PDS over the hook plate relies on the fact that there is no need for a second operation for removing the implant [17].

Anatomical reconstruction of the CC and AC ligaments

Several biomechanical studies have demonstrated the superiority of anatomical reconstructions over other procedures with regard to the potential to emulate the properties of the native ligaments [21].

Carofino and Mazzocca developed a reconstructive technique that involves a tendon graft fixation in the native locations of the CC ligaments [22]. They performed clavicular tunnels and placed the graft in a figure-of-eight fashion, which was fixed with interferential bio-tenodesis screws [22]. The authors proposed a subcoracoid pass of the tendon graft (without coracoid tunnel), which finally rises from the coracoid to the clavicle; both ends of the graft cross between them to form the above-mentioned configuration. In a series of 106 cases with a mean follow-up of 21 months, they reported a significant improvement of the preoperative clinical results [22].

Yoo et al. described the anatomical reconstruction of the CC ligaments assisted by arthroscopy, in which three bone tunnels were performed in the native origins of the CC ligaments—two tunnels in the clavicle and one in the coracoid [23]. The authors argue that making only one tunnel in the coracoid carries a low risk of iatrogenic fracture. The described technique does not involve the use of a primary mechanical stabilizer that would protect the graft during the integration process to the bone tunnels; a reason why it can be inferred that their reconstructions may be prone to distraction forces that might affect the initially obtained ACJ reduction. In fact, although the authors report satisfactory clinical results, subtle secondary displacements were observed at final follow-up in 100% of patients in their series (13/13) [23].

In a study by Natera et al., the senior author (Dr. Sarasquete) added a CC suspension device to the anatomical reconstruction of the CC ligaments with a tendon allograft [7] in order to improve the primary mechanical fixation and thus protect the tendon graft during the integration process to the bone tunnels and reduce the rate of secondary vertical displacements, Likewise, the study group led by the above-mentioned author described the use of two suspension devices with two tunnels in the coracoid, a technique that in the acute setting would provide greater resistance to vertical translation [24]. A summary of the main aspects of the cited biomechanical studies is shown in Table 2.

Table 2 Summary of the main aspects of the cited biomechanical studies

Synthetic grafts

The use of synthetic ligament reconstructions is an option that could be considered for the treatment of chronic ACJ instability. The synthetic grafts most commonly used are the Ligament Advanced Reinforcement System (LARS®; Surgical Implants and Devices, Arc-sur-Tille, France), the Dacron® graft and the Ligastic® [25, 26]. Several authors reported satisfactory clinical results with the LARS® [34], and unsatisfactory results with the Dacron® [25] and the Ligastic® [26]. With regard to the Dacron® vascular prostheses, Fraschini et al. reported a complication rate of 43.3% (13/30 patients), in which 23.3% (7/30 patients) had a graft tear [25]. Regarding the LARS®, the rate of graft tears described by the authors was 3.3% (1/30 patients) [25].

Regarding the Ligastic®, Mares et al. described a rate of clavicular osteolysis of 22% (6/27 patients) [26]. In fact, these authors reported in their study that they are currently rejecting the use of this type of implant, and advising against its use. However, further studies are needed to clarify the role of synthetic grafts in the management of chronic ACJ injuries.

Muccioli et al. compared the outcomes of ACJ reconstruction with the LARS® in professional athletes with non-professional athletes at a 2-year minimum follow-up. They found that all clinical (Oxford and Constant) scores, as well as patient satisfaction, improved significantly from preoperative to follow-up intervals, with no differences between groups, and only 2% of failures (re-dislocations) [27]. On the other hand, Fauci et al. compared the clinical and radiographic outcomes of ACJ stabilization performed in patients with chronic ACJ dislocation using a biological allograft or a synthetic ligament, and reported that the ʽbiological’ group achieved significantly better clinical scores than the 'synthetic' group, at both 1- and 4-year follow-up. The authors concluded that the biological graft afforded better clinical and radiographic outcomes than the synthetic ligament in patients with chronic ACJ instability [28].

Dynamic stabilization of the ACJ

An osteotomy is made to the coracoid process, which is later transferred to the inferior aspect of the clavicle with the attached conjoined tendon [29]. The bone block is fixed to the clavicle by means of a screw with a spike washer. In this way, the conjoined tendon is converted to a depressor of the clavicle. This concept does not directly address the pathomechanics of an ACJ injury in which, rather than a superior displacement of the clavicle, it is the scapula that descends [1]. Despite this issue, the technique has been used in both the acute and chronic settings with satisfactory results [30].

Distal third clavicle excision

Excision of the distal third of the clavicle (Mumford procedure) may represent a solution to a painful chronic ACJ injury (grade I–III) [31]. Osteoarthritic changes have been described to be mostly restricted to type I and type II injuries, since the greater separation of the bone ends in higher-grade injuries may prevent the development of this complication [31]. However, degenerative changes in the articular disc and lateral end of the clavicle may be found during surgery and might be a source of pain in high-grade injuries. This technique must involve the resection of only 5 mm of the distal third of the clavicle, since (in cases of ACJ injuries grade I–II) the trapezoid ligament is only 2.5 cm medial to the distal end of the clavicle [4]; more generous resections could affect the clavicular insertion of the trapezoid ligament.

Authors preferred technique

This technique has been previously described [7].

We perform an arthroscopy-assisted reconstruction in order to be able to diagnose and treat possible associated glenohumeral injuries (Fig. 2). We propose anatomical reconstruction of the CC ligaments using a semitendinosus tendon allograft (Fig. 3a, b). In Fig. 3c, the radiological aspect of a right shoulder in which this technique was performed can be appreciated. In a contact player, we prefer to use a tendon autograft, which may be the ipsilateral palmaris longus.

Fig. 2
figure2

a Anterolateral perspective of a right shoulder positioned in the operating room, with a history of a chronic grade V ACJ injury. b Biceps-labrum complex viewed from the posterior portal. Notice the degenerative aspect of the biceps insertion, which indicates an associated glenohumeral injury

Fig. 3
figure3

a Semitendinous allograft after being sutured with a metal-core suture in both of its limbs. b Both limbs of the graft coming out of the clavicle once fixed in both tunnels with bio-tenodesis interference screws. The ZipTight is tied by threading the sliding suture in the washer. c AP X-ray of a right shoulder in which an anatomical reconstruction of CC ligaments with tendon allograft was performed in the chronic setting. Observe the trapezoid tunnel in the clavicle, lateral to the conoid tunnel in the clavicle, through which also passes the suspension device

The technique implies one tunnel at the coracoid, and two tunnels at the clavicle. These tunnels aim to emulate the anatomical locations of the CC ligaments. We also add a CC suspension device in order to guarantee primary stability of the reconstruction.

A subacromial approach to the base of the coracoid is performed in association with a Mumford procedure. A transverse skin incision over the clavicle is performed. The conoid native insertion is 4.5 cm medial to the distal end of the clavicle and the trapezoid native insertion is 2.5 cm and subtly anterior when compared to the conoid [4].

A cross section of the deltotrapezial fascia is performed, and the AC drilling guide is placed at the base of the coracoid with the sliding tube at the superior aspect of the clavicle, 4.5 cm medial to its distal end (conoid native origin) (Fig. 4a). A K-wire is passed followed by the cannulated drill. The K-wire is removed and the cannulated drill is maintained in the same position (Fig. 4b). Subsequently, the same procedure should be performed for the clavicular tunnel of the trapezoid ligament. Shuttle sutures are passed through the cannulated drills (Fig. 4c). Two metal-core sutures are tied to the distal end of the shuttle suture that passes through the coracoid. A superior perspective of the clavicle shows both shuttle sutures emerging from the tunnels (Fig. 4d).

Fig. 4
figure4

Reproduced with permission and copyright© of Arthroscopy Techniques, Elsevier. a The AC drilling guide is placed at the coracoid base with the sliding tube of the guide in the superior aspect of the clavicle, 4.5 cm medial to its lateral border (conoid native origin). A 2.4-mm K-wire is passed through the AC guide. b A cannulated 4.5- to 6-mm (depending on the graft diameter) drill is passed over the K-wire and comes out from the inferior aspect of the coracoid. c A shuttle 1-mm PDS suture is passed through the cannulated drill located in the trapezoid tunnel. The PDS is recovered with a grasper from the anterior portal. d Superior perspective of the clavicle in which both shuttle sutures are emerging from the tunnels. e The PDS that arises from the trapezoid tunnel in the clavicle is pulled out in a cranial direction to recover the limb of the graft that is going to surround the base of the coracoid at its lateral aspect, coming from its tunnel and then being directed laterally and superiorly, configuring the anatomical 'V' shape of the graft. f Once the graft has passed through both clavicle tunnels, the ZipTight is tied to the distal limb of the shuttle FiberWire that is still free in the conoid tunnel

One of the metal-core sutures passing through the conoid tunnel is temporarily tied to one of the ends of the tendon graft. The other end of the graft is temporarily tied to the shuttle suture, which is coming out of the trapezoid clavicle and exits through the anterior portal.

The graft is passed by means of pulling cranially on the metal-core suture that comes out of the conoid tunnel. Subsequently, the shuttle suture which is coming out of the trapezoid clavicle tunnel is pulled in a superior direction; the graft is directed laterally and superiorly, conforming to the anatomical 'V' configuration of the reconstruction (Fig. 4e).

One of the ends of the shuttle metal-core suture is still free in the conoid tunnel. This suture is now tied to the CC suspension device, and pulled out in a cranial direction so the device passes in a retrograde direction (Fig. 4f).

The graft is fixed in the clavicular portion of the tunnels with bio-tenodesis interferential screws (Fig. 5a). The washer should be threaded with the sliding sutures, in order to be able to bring it down until it is applied over the clavicle (Fig. 5b). The assistants must reduce the ACJ by pushing the elbow upwards and the clavicle downwards at the same time. The CC suspension device is now locked (Fig. 5c). Both limbs of the graft are crossed over each other and sutured to themselves (Fig. 5d). The remaining graft is sectioned and removed. The deltotrapezial fascia is carefully reconstructed.

Fig. 5
figure5

a Before the ZipTight is tensioned, the graft should be fixed in the clavicular portion of the conoid tunnel with a 4.5- to 5.5-mm (same diameter of the tunnel) bio-tenodesis interference screw. Reproduced with permission and copyright© of Arthroscopy Techniques, Elsevier. b Both limbs of the graft coming out of the clavicle when fixed in both tunnels with bio-tenodesis interference screws. The ZipTight is tied by threading the sliding suture in the washer. To avoid any harm to the sutures of the ZipTight with the screw, the graft should be placed in an intermediate position between the screw and the sutures. c The ZipTight has been tied by pulling alternatively on both limbs of the blue traction sutures in a cranial direction to make the washer go down until it touches the clavicle and self-locks, providing mechanical stabilization of the reconstruction. d Both limbs of the graft are crossed over each other and sutured to themselves. The remnant of the graft is sectioned and removed

The described technique provides the advantages of minimally invasive surgery, avoids the biomechanical disadvantages related to rigid metal hardware procedures, offers greater biomechanical resistance thus minimizing the risk of secondary displacements related to non-anatomical techniques, and combines primary mechanical stabilization and definitive biological stabilization represented by the graft, once integrated to the bone (Fig. 6a, b).

Fig. 6
figure6

a Final arthroscopic view from the lateral portal. The graft is coming out of the coracoid tunnel, ascending toward the trapezoid tunnel in the clavicle. The flip of the ZipTight is supported in the inferior aspect of the coracoid. b Final anatomical 'V' configuration of the CC reconstruction, with the flip of the ZipTight supported in the inferior aspect of the coracoid and both limbs of the graft are crossed over each other and sutured to themselves. Reproduced with permission and copyright© of Arthroscopy Techniques, Elsevier

The results obtained with this technique have been published previously [32]. Ten patients with a mean age of 41 years underwent surgery after failure of conservative measures. The clinical outcomes showed a significant improvement from the visit prior to surgery to the last follow-up in all patients, and no secondary vertical instability was registered in any of the cases [32].

Fixation method of the tendinous allograft in the coracoid process

It has been reported that suture subcoracoid loops tend to dislocate anteriorly due to the ascending slope that is represented by the most caudal portion of the base of the coracoid [33]. It has also been shown that the use of subcoracoid suture loops can involve a shear deleterious effect on the bone [34].

Other authors propose techniques that do not involve making tunnels at the coracoid, but pass the graft around the caudal portion of the bone. We think that by taking into consideration the fact that there is no contact between the cancellous bone and the collagen of the tendon graft [22], integration of the graft might not be developed.

Postoperative management

Regardless of the chosen technique, due to the fact that biological augmentation should be employed in the chronic setting, there should be a protection period of the reconstruction in order to guarantee integration of the graft to the bone tunnels [7].

The shoulder should be maintained in a sling for 46 weeks. Patients should be allowed from the beginning to fully and actively move the elbow, wrist, and hand and should be allowed to passively move the shoulder into no more than 90° of elevation in the plane of the scapula. The exercise program should be started after the sixth week. Pendulum exercises must begin in the fourth week, and active range of motion is allowed from the sixth week onwards. Exercises to regain strength are initiated once the patient achieves full, pain-free passive and active range of motion. These exercises are primarily directed toward scapular stabilization. Return to work without restrictions is allowed at 12–16 weeks after surgery, and contact sports, as well as tasks requiring major efforts should be avoided for 4–6 months after surgery. The achievement of a full recovery and the return to maximum strength and function can take from 9−12 months.

Complications

The profile of complications that can be expected after surgery for ACJ instability depends on whether the reconstruction is performed in the acute or chronic setting, on the type of fixation used, and on whether the reconstruction is performed using arthroscopy-assisted or open surgery. The rate of complications according to different studies is shown in Table 3.

Table 3 Rate of complications according to different studies

With regard to infection rates, a systematic review of the literature reports that the overall rate of superficial infections is approximately 3.8% for arthroscopic procedures [35], in contrast to a rate of up to 5% for procedures performed by open surgery [35], and up to 8% in those procedures in which a tendon graft was used [36, 37].

The failure rate after fixation in the chronic setting using only a tendon graft, has been reported to be approximately ≥50% [35, 38], while the failure rate after management in the acute setting has been reported to be approximately 26.8% [35].

It has been reported that these differences may be due to the fact that the tendon graft tends to lengthen over time, and it may also emulate a 'windshield' effect at the level of the clavicular tunnels, a situation that eventually ends with widening of the tunnels [39].

Regarding the incidence of fractures of the coracoid process, it has been reported that the overall rate (both mono-tunnel and double-tunnel techniques) is approximately 5.3% [35].

Non-surgical management of chronic ACJ instability

Gumina et al. reported that the prevalence of scapular dyskinesis (Fig. 7) in patients with chronic ACJ instability (Rockwood grade III) can be up to 70.6% [40], and that the prevalence of SICK scapula [41] (Scapular malposition, Inferior medial border prominence, Coracoid pain and malposition, and dyskinesis of scapular movement) can be up to 58.3% [40]. This group of patients might develop persistent shoulder pain that could make them unable to return to their previous daily life activities [42]. The occurrence of modifications in the scapular orientation leads to cinematic alterations of the muscles, thus perturbing the shoulder girdle biomechanics. Likewise, it has been shown that the prevalence of scapular dyskinesis in those patients managed surgically is lower when compared to patients managed non-surgically [2, 40].

Fig. 7
figure7

a and b Posterior perspective of two patients performing shoulder forward flexion. Notice that the inferomedial border of the right scapula (red arrows) shows a prominence. These two patients had a history of chronic unstable ACJ injuries that were conservatively treated

Patients with this syndrome may refer shoulder pain at the ACJ and at the coracoid, posterior shoulder pain sometimes irradiated to the cervical paraspinal region and to the lateral aspect of the arm, or even radicular symptoms.

Carbone et al. proposed a rehabilitation protocol for patients with scapular dyskinesis [43]. The protocol consists of 12 exercises aimed to strength the scapular muscles. These authors described a series of 24 patients with a history of chronic ACJ instability (grade III) in which 100% (24/24) had scapular dyskinesis and 58.33% (14/24) had SICK scapula [43]. Twelve months after having accomplished the rehabilitation protocol, 21.73% (5/23) of the patients still had scapular dyskinesis and 17.4% (4/23) still had SICK scapula. They concluded that scapular dyskinesis and SICK scapula secondary to chronic ACJ instability might show improvement within 6 weeks of starting this rehabilitation protocol.

Discussion

Arthroscopy-assisted surgery versus open surgery

With regard to the advantages that arthroscopy-assisted surgery may offer over open surgery in cases of chronic ACJ instability, it is important to mention that associated glenohumeral lesions can be diagnosed and treated [7]. Some authors have reported that the incidence of lesions associated with unstable ACJ injuries can be up to 30% [44]. In the management of chronic ACJ instability, it is important to guarantee that there is no interposition of the deltotrapezial fascia between the clavicle and the acromion, a situation that can only be accomplished by means of making a mini-approach just above the ACJ. Once anatomical reduction of the ACJ has been reached, the deltotrapezial fascia should be carefully reconstructed in order to ensure adequate vertical and horizontal stability [7].

Anatomical versus non-anatomical reconstructions

Anatomical AC and CC ligament reconstruction techniques have become increasingly popular. Several clinical and biomechanical studies have shown their superiority in reproducing the strength and stiffness of the native ACJ complex when compared to other reconstructive techniques [20, 36, 45]. Biomechanical studies of ACJ reconstructions with free-tissue grafts for both the CC and the AC ligamentous complex have shown that these techniques provide ACJ stability similar to that of the native ACJ [45]. Likewise, it is currently clear that by taking into consideration the biomechanics and the resistance of the reconstruction that anatomical procedures are superior techniques when compared to the classical Weaver–Dunn technique [45].

Lafosse et al. describe an arthroscopic technique indicated for cases of chronic ACJ instability, in which they propose CA ligament transfer in order to reproduce the function of the torn CC ligaments [8]. It has been reported that transposition of the CA ligament of the Weaver–Dunn technique offers a lower resistance to vertical translation than anatomical CC reconstructions with tendon grafts [20].

LaPrade et al. described an open non-anatomical technique in which they propose the use of a semitendinosus allograft, which passes through a tunnel in the clavicle and another in the coracoid [9]. This technique entails a biomechanical disadvantage that does not take into account the anatomical location of the CC ligaments [9]. The authors recognize that in some patients, an elongation of the graft may be developed, a situation that may result in persistent ACJ instability in the vertical plane [9].

In a prospective, comparative, clinical study, Tauber et al. showed that anatomical ligament reconstruction of the conoid and trapezoid ligaments with tendon grafts results in superior outcomes compared to the modified Weaver−Dunn technique [36].

Anteroposterior (AP) stabilization

The shoulder community has shown an increasing interest in anatomical CC ligament reconstruction, because these concepts aim to recreate the force vectors of both the conoid and trapezoid ligaments, thus restoring both horizontal and vertical instability. Despite the recent development of numerous reconstructive techniques, residual AP post-surgical instability remains a matter of concern [46]. Likewise, the importance of simultaneous reconstruction of the AC ligaments has been widely studied and demonstrated [47]. It has been reported that patients who underwent surgery for unstable ACJ injury, and show remaining AP post-surgical instability, may have significantly inferior clinical results [48]. Likewise, it has been also reported that persistent AP post-surgical instability is the only factor that may adversely affect the clinical outcomes [48]. For this reason, reconstructive strategies must give the same importance to AC reconstruction as to CC reconstruction [49].

Arthroscopic approach to the coracoid process

Some authors propose a direct skin incision over the tip of the coracoid, blunt dissection and identification of its base, in order to place the drilling guide [50]. These techniques are performed in a 'blind' manner, and therefore lack the precision that direct visualization may provide. To guarantee a proper view of the lower portion of the base of the coracoid, several arthroscopic techniques that facilitate the process of tunnel-making and implant-positioning have been described [79]. Glenohumeral access involves the need to release the superior and middle glenohumeral ligaments, in order to gain access to the coracoid process [51]. On the other hand, subacromial access to the coracoid has the advantage over glenohumeral access, as it does not involve the potential deleterious effect that may result from the release of the superior and middle glenohumeral ligaments [7].

Overview

Considering all the procedures described in this review, patients with shoulder symptoms resulting from chronic ACJ instability may benefit from surgical treatment. The procedures considered for the management of chronic ACJ instability should take into account the biological aspects; for this reason the use of either a tendon graft, ligament or osteotendinous transposition should always be considered. Likewise, the fundamental role that primary mechanical fixation may play should to be taken into account, in order to protect the integration period of biological augmentation to the bone.

References

  1. 1.

    Mazzocca AD, Arciero RA, Bicos J (2007) Evaluation and treatment of acromioclavicular joint injuries. Am J Sports Med 35:316–329. doi:10.1177/0363546506298022

  2. 2.

    Murena L, Canton G, Vulcano E, Cherubino P (2013) Scapular dyskinesis and SICK scapula syndrome following surgical treatment of type III acute acromioclavicular dislocations. Knee Surg Sports Traumatol Arthrosc 21:1146–1150

  3. 3.

    Lee SJ, Nicholas SJ, Akizuki KH, McHugh MP, Kremenic IJ, Ben- Avi S (2003) Reconstruction of the coracoclavicular ligaments with tendon grafts: a comparative biomechanical study. Am J Sports Med 31:648–654

  4. 4.

    Rios CG, Arciero RA, Mazzocca AD (2007) Anatomy of the clavicle and coracoid process for reconstruction of the coracoclavicular ligaments. Am J Sports Med 35:811–817

  5. 5.

    Gstettner C, Tauber M, Hitzl W, Resch H (2008) Rockwood type III acromioclavicular dislocation: surgical versus conservative treatment. J Shoulder Elbow Surg 17:220–225

  6. 6.

    Weinstein DM, McCann PD, McIlveen SJ, Flatow EL, Bigliani LU (1995) Surgical treatment of complete acromioclavicular dislocations. Am J Sports Med 23:324–331

  7. 7.

    Natera L, Sarasquete Reiriz J, Abat F (2014) Anatomic reconstruction of chronic coracoclavicular ligament tears: arthroscopic-assisted approach with nonrigid mechanical fixation and graft augmentation. Arthrosc Tech 15;3(5):e583-8. doi:10.1016/j.eats.2014.06.014. eCollection 2014 Oct

  8. 8.

    Lafosse L, Baier GP, Leuzinger J (2005) Arthroscopic treatment of acute and chronic acromioclavicular joint dislocation. Arthroscopy 21:1017

  9. 9.

    LaPrade RF, Hilger B (2005) Coracoclavicular ligament reconstruction using a semitendinosus graft for failed acromioclavicular separation surgery. Arthroscopy 21:1277.e1-1277.e5

  10. 10.

    Beitzel K, Mazzocca AD, Bak K, Itoi E, Kibler WB, Mirzayan R, Imhoff AB, Calvo E, Arce G, Shea K, Upper Extremity Committee of ISAKOS (2014) ISAKOS upper extremity committee consensus statement on the need for diversification of the Rockwood classification for acromioclavicular joint injuries. Arthroscopy 30(2):271–278. doi:10.1016/j.arthro.2013.11.005

  11. 11.

    Rolf O, Hann von Weyhern A, Ewers A, Boehm TD, Gohlke F (2008) Acromioclavicular dislocation Rockwood III–V: results of early versus delayed surgical treatment. Arch Orthop Trauma Surg 128:1153–1157

  12. 12.

    Mignani G, Rotini R, Olmi R, Marchiodi L, Veronesi CA (2002) The surgical treatment of Rockwood grade III acromio-clavicular dislocations. Chir Organi Mov 87:153–161

  13. 13.

    Dumontier C, Sautet A, Man M, Apoil A (1995) Acromioclavicular dislocations: treatment by coracoacromial ligamentoplasty. J Shoulder Elbow Surg 4:130–134

  14. 14.

    von Heideken J, Boström Windhamre H, Une-Larsson V, Ekelund A (2013) Acute surgical treatment of acromioclavicular dislocation type V with a hook plate: superiority to late reconstruction. J Shoulder Elbow Surg 22:9–17

  15. 15.

    Boileau P, Old J, Gastaud O, Brassart N, Roussanne Y (2010) All-arthroscopic Weaver–Dunn–Chuinard procedure with double-button fixation for chronic acromioclavicular joint dislocation. Arthroscopy 26(2):149–160. doi:10.1016/j.arthro.2009.08.008

  16. 16.

    Weaver JK, Dunn HK (1972) Treatment of acromioclavicular injuries, especially complete acromioclavicular separation. J Bone Joint Surg Am 54-A:1187–1194

  17. 17.

    Boström Windhamre HA, von Heideken JP, Une-Larsson VE, Ekelund AL (2010) Surgical treatment of chronic acromioclavicular dislocations: a comparative study of Weaver-Dunn augmented with PDS-braid or hook plate. J Shoulder Elbow Surg 19:1040–1048

  18. 18.

    Shin SJ, Yun YH, Yoo JD (2009) Coracoclavicular ligament reconstruction for acromioclavicular dislocation using 2 suture anchors and coracoacromial ligament transfer. Am J Sports Med 37:346–351

  19. 19.

    Liu HH, Chou YJ, Chen CH, Chia WT, Wong CY (2010) Surgical treatment of acute acromioclavicular joint injuries using a modified Weaver-Dunn procedure and clavicular hook plate. Orthopedics 11;33(8). doi:10.3928/01477447-20100625-10

  20. 20.

    Grutter PW, Petersen SA (2005) Anatomical acromioclavicular ligament reconstruction: a biomechanical comparison of reconstructive techniques of the acromioclavicular joint. Am J Sports Med 33:1723–1728. doi:10.1177/0363546505275646

  21. 21.

    Deshmukh AV, Wilson DR, Zilberfarb JL, Perlmutter GS (2004) Stability of acromioclavicular joint reconstruction: biomechanical testing of various surgical techniques in a cadaveric model. Am J Sports Med 32:1492–1498. doi:10.1177/0363546504263699

  22. 22.

    Carofino BC, Mazzocca AD (2010) The anatomic coracoclavicular ligament reconstruction: surgical technique and indications. J Shoulder Elbow Surg 19:37–46. doi:10.1016/j.jse.2010.01.004

  23. 23.

    Yoo YS, Seo YJ, Noh KC, Patro BP, Kim DY (2011) Arthroscopically assisted anatomical coracoclavicular ligament reconstruction using tendon graft. Int Orthop 35(7):1025–1030. doi:10.1007/s00264-010-1124-3

  24. 24.

    Abat F, Sarasquete J, Natera LG, Calvo Á, Pérez-España M, Zurita N, Ferrer J, Del Real JC, Paz-Jimenez E, Forriol F (2015) Biomechanical analysis of acromioclavicular joint dislocation repair using coracoclavicular suspension devices in two different configurations. J Orthop Traumatol 16(3):215–219. doi:10.1007/s10195-015-0346-y

  25. 25.

    Fraschini G, Ciampi P, Scotti C, Ballis R, Peretti GM (2010) Surgical treatment of chronic acromioclavicular dislocation: comparison between two surgical procedures for anatomic reconstruction. Injury 41:1103–1106

  26. 26.

    Mares O, Luneau S, Staquet V, Beltrand E, Bousquet PJ, Maynou C (2010) Acute grade III and IV acromioclavicular dislocations: outcomes and pitfalls of reconstruction procedures using a synthetic ligament. Orthop Traumatol Surg Res 96:721–726

  27. 27.

    Marcheggiani Muccioli GM, Manning C, Wright P, Grassi A, Zaffagnini S, Funk L. (2014) Acromioclavicular joint reconstruction with the LARS ligament in professional versus non-professional athletes. Knee Surg Sports Traumatol Arthrosc. doi:10.1007/s00167-014-3231-y (Epub ahead of print)

  28. 28.

    Fauci F, Merolla G, Paladini P, Campi F, Porcellini G (2013) Surgical treatment of chronic acromioclavicular dislocation with biologic graft vs synthetic ligament: a prospective randomized comparative study. J Orthop Traumatol 14(4):283–290

  29. 29.

    Berson BL, Gilbert MS, Green S (1978) Acromioclavicular dislocations: treatment by transfer of the conjoined tendon and distal end of the coracoid process to the clavicle. Clin Orthop Relat Res 135:157e64

  30. 30.

    Bailey RW, Metten CF, O’Connor GA, Titus PD, Baril JD, Moosman DA (1976) A dynamic method of repair for acute and chronic acromioclavicular disruption. Am J Sports Med 4:58e71

  31. 31.

    Snyder SJ, Banas MP, Karzel RP (1995) The arthroscopic Mumford procedure: an analysis of results. Arthroscopy 11(2):157–164

  32. 32.

    Natera-Cisneros L, Santiago-Boccolini H, Sarasquete-Reiriz J (2015) Treatment of chronic acromioclavicular joint instability. Acta Ortop Mex 29(3):164–171

  33. 33.

    Jerosch J, Filler T, Peuker E, Greig M, Siewering U (1999) Which stabilization technique corrects anatomy best in patients with AC-separation? An experimental study. Knee Surg Sports Traumatol Arthrosc 7:365–372

  34. 34.

    Guttmann D, Paksima NE, Zuckerman JD (2000) Complications of treatment of complete acromioclavicular joint dislocations. Instr Course Lect 49:407–413

  35. 35.

    Woodmass JM, Esposito JG, Ono Y, Nelson AA, Boorman RS, Thornton GM, Lo IK (2015) Complications following arthroscopic fixation of acromioclavicular separations: a systematic review of the literature. Open Access J Sports Med 6:97–107. doi:10.2147/OAJSM.S73211

  36. 36.

    Tauber M, Gordon K, Koller H, Fox M, Resch H (2009) Semitendinosus tendon graft versus a modified Weaver–Dunn procedure for acromioclavicular joint reconstruction in chronic cases: a prospective comparative study. Am J Sports Med 37:181–190. doi:10.1177/0363546508323255

  37. 37.

    Modi CS, Beazley J, Zywiel MG, Lawrence TM, Veillette CJ (2013) Controversies relating to the management of acromioclavicular joint dislocations. Bone Joint J 95-B(12):1595–1602

  38. 38.

    Milewski MD, Tompkins M, Giugale JM, Carson EW, Miller MD, Diduch DR (2012) Complications related to anatomic reconstruction of the coracoclavicular ligaments. Am J Sports Med 40(7):1628–1634

  39. 39.

    Cook JB, Shaha JS, Rowles DJ, Bottoni CR, Shaha SH, Tokish JM (2012) Early failures with single clavicular transosseous coracoclavicular ligament reconstruction. J Shoulder Elbow Surg 21(12):1746–1752

  40. 40.

    Gumina S, Carbone S, Postacchini F (2009) Scapular dyskinesis and SICK scapula syndrome in patients with chronic type III acromioclavicular dislocation. Arthroscopy 25(1):40–45. doi:10.1016/j.arthro.2008.08.019

  41. 41.

    Burkhart SS, Morgan CD, Kibler W (2003) The disabled throwing shoulder: spectrum of pathology. Part III: the SICK scapula, scapular dyskinesis, the kinetic chain, and rehabilitation. Arthroscopy 19:641–661

  42. 42.

    Beitzel K, Cote MP, Apostolakos J, Solovyova O, Judson CH, Ziegler CG, Edgar CM, Imhoff AB, Arciero RA, Mazzocca AD (2013) Current concepts in the treatment of acromioclavicular joint dislocations. Arthroscopy 29:387–397. doi:10.1016/j.arthro.2012.11.023

  43. 43.

    Carbone S, Postacchini R, Gumina S (2015) Scapular dyskinesis and SICK syndrome in patients with a chronic type III acromioclavicular dislocation. Results of rehabilitation. Knee Surg Sports Traumatol Arthrosc 23(5):1473–1480. doi:10.1007/s00167-014-2844-5

  44. 44.

    Pauly S, Kraus N, Greiner S, Scheibel M (2013) Prevalence and pattern of glenohumeral injuries among acute high-grade acromioclavicular joint instabilities. J Shoulder Elbow Surg 22:760–766. doi:10.1016/j.jse.2012.08.016

  45. 45.

    Michlitsch MG, Adamson GJ, Pink M, Estess A, Shankwiler JA, Lee TQ (2010) Biomechanical comparison of a modified Weaver-Dunn and a free-tissue graft reconstruction of the acromioclavicular joint complex. Am J Sports Med 38:1196–1203. doi:10.1177/0363546509361160

  46. 46.

    Wellmann M, da Silva G, Lichtenberg S, Magosch P, Habermeyer P (2013) Instability pattern of acromioclavicular joint dislocations type Rockwood III: relevance of horizontal instability. Orthopade 42:271–277. doi:10.1007/s00132-013-2085-1

  47. 47.

    Dawson PA, Adamson GJ, Pink MM, Kornswiet M, Lin S, Shankwiler JA, Lee TQ (2009) Relative contribution of acromioclavicular joint capsule and coracoclavicular ligaments to acromioclavicular stability. J Shoulder Elbow Surg 18:237–244. doi:10.1016/j.jse.2008.08.003

  48. 48.

    Scheibel M, Dröschel S, Gerhardt C, Kraus N (2011) Arthroscopically assisted stabilization of acute high-grade acromioclavicular joint separations. Am J Sports Med 39(7):1507–1516

  49. 49.

    Beitzel K, Mazzocca AD (2014) Open anatomic reconstruction of chronic acromioclavicular instability. Oper Orthop Traumatol 26(3):237–244. doi:10.1007/s00064-013-0277-9 (Epub 2014 Jun 14)

  50. 50.

    Wellmann M, Zantop T, Petersen W (2007) Minimally invasive coracoclavicular ligament augmentation with a flip button/polydioxanone repair for treatment of total acromioclavicular joint dislocation. Arthroscopy 23:1132.e1-5

  51. 51.

    Baumgarten KM, Altchek DW, Cordasco FA (2006) Arthroscopically assisted acromioclavicular joint reconstruction. Arthroscopy 22:228.e1–228.e6

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Correspondence to Luis Natera Cisneros.

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Keywords

  • Unstable acromioclavicular joint injuries
  • Chronic setting
  • Arthroscopically assisted management
  • Anatomical ligament reconstruction
  • Coracoclavicular ligaments
  • Scapular dyskinesis